dc.contributor.author | Jacobsen, Henrik Børsting | |
dc.contributor.author | Bjørngaard, Johan Håkon | |
dc.contributor.author | Hara, Karen Walseth | |
dc.contributor.author | Borchgrevink, Petter Chr. | |
dc.contributor.author | Woodhouse, Astrid | |
dc.contributor.author | Landrø, Nils Inge | |
dc.contributor.author | Harris, Anette | |
dc.contributor.author | Stiles, Tore C. | |
dc.date.accessioned | 2015-01-06T12:07:25Z | |
dc.date.available | 2015-01-06T12:07:25Z | |
dc.date.issued | 2014-05-02 | eng |
dc.identifier.issn | 1932-6203 | |
dc.identifier.uri | https://hdl.handle.net/1956/9077 | |
dc.description.abstract | Objective: This study aimed to (1) See whether increased or decreased variation relate to subjective reports of common somatic and psychological symptoms for a population on long-term sick leave; and (2) See if this pattern in variation is correlated with autonomic activation and psychological appraisal. Methods: Our participants (n = 87) were referred to a 3.5-week return-to-work rehabilitation program, and had been on paid sick leave .8 weeks due to musculoskeletal pain, fatigue and/or common mental disorders. An extensive survey was completed, addressing socio-demographics, somatic and psychological complaints. In addition, a physician and a psychologist examined the participants, determining baseline heart rate, medication use and SCID-I diagnoses. During the 3.5-week program, the participants completed the Trier Social Stress Test for Groups. Participants wore heart rate monitors and filled out Visual Analogue Scales during the TSST-G. Results: Our participants presented a low cortisol variation, with mixed model analyses showing a maximal increase in free saliva cortisol of 26% (95% CI, 0.21–0.32). Simultaneously, the increase in heart rate and Visual Analogue Scales was substantial, indicating autonomic and psychological activation consistent with intense stress from the Trier Social Stress Test for Groups. Conclusions: The current findings are the first description of a blunted cortisol response in a heterogeneous group of patients on long-term sick leave. The results suggest lack of cortisol reactivity as a possible biological link involved in the pathway between stress, sustained activation and long-term sick leave. | en_US |
dc.language.iso | eng | eng |
dc.publisher | PLOS | eng |
dc.rights | Attribution CC BY | eng |
dc.rights.uri | http://creativecommons.org/licenses/by/4.0 | eng |
dc.title | The role of stress in absenteeism: Cortisol responsiveness among patients on long-term sick leave. | eng |
dc.type | Peer reviewed | |
dc.type | Journal article | |
dc.date.updated | 2015-01-06T12:02:02Z | |
dc.description.version | publishedVersion | |
dc.rights.holder | Copyright 2014 2014 Jacobsen et al | |
dc.source.articlenumber | e96048 | |
dc.identifier.doi | https://doi.org/10.1371/journal.pone.0096048 | |
dc.identifier.cristin | 1140155 | |
dc.source.journal | PLoS ONE | |
dc.source.40 | 9 | |
dc.source.14 | 5 | |